NURS 6501 NURS6501 NURS/6501 Wk 2a discussion – Inflammatory bowel disease
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NURS 6501 NURS6501 NURS/6501 Wk 2a discussion – Inflammatory bowel disease
Inflammatory bowel disease Pathophysiology
Inflammatory bowel disease (IBD) is depicted by chronic deteriorating intestinal inflammation. IBD comprises Crohn’s disease (CD) and ulcerative colitis (UC), which are distinct chronic bowel-relapsing inflammatory disorders (Zhang & Li, 2014).
Current research specified that the individual’s genetic receptiveness, external environment, intestinal microbial flora, and immune responses altogether are involved and functionally incorporated in the pathogenesis of IBD (Zhang & Li, 2014).
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NURS 6501 NURS6501 NURS/6501 Wk 2a discussion – Inflammatory bowel disease
Inflammatory bowel disease Pathophysiology
Inflammatory bowel disease (IBD) is depicted by chronic deteriorating intestinal inflammation. IBD comprises Crohn’s disease (CD) and ulcerative colitis (UC), which are distinct chronic bowel-relapsing inflammatory disorders (Zhang & Li, 2014).
Current research specified that the individual’s genetic receptiveness, external environment, intestinal microbial flora, and immune responses altogether are involved and functionally incorporated in the pathogenesis of IBD (Zhang & Li, 2014).
Chron’s Disease can begin transmural inflammation and affect any part of the gastrointestinal tract, usually the terminal ileum or the perianal region, in a non-continuous type (Zhang & Li, 2014). Unlike Ulcerative Colitis, Chron’s Disease is oftenconnected with impediments such as abscesses, fistulas and strictures.
While in comparison, Ulcerative Colitis is characterized by mucosal inflammation and limited to the colon (Zhang & Li, 2014).The intestinal tract where IBD occurs is vital to the immune system, and the innate and the adaptive immune systems are balanced in multifaceted interactions with intestinal microbes under homeostatic conditions (Kim & Cheon, 2017).
Nevertheless, for several purposes, idiopathic intestinal inflammations such as inflammatory bowel disease can occur when this homeostasis is interrupted (Kim & Cheon, 2017).
Conventionally, it has been thought that Th1 cells play a significant function in pathogenesis associated to the chronicity of intestinal inflammation, particularly in Chron’s Disease, while Th2 cells have been believed to play an imperative role in Ulcerative Colitis (Kim & Cheon, 2017).
Lately, conversely, the triggering of Th17 cells and imbalance of “Th17/regulatory T (Treg) cells” are accepted to be an essential factor in the progress of intestinal inflammation (Kim & Cheon, 2017)
NURS 6501 NURS6501 NURS/6501 Wk 2a discussion – Inflammatory bowel disease
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NURS 6501 NURS6501 NURS/6501 Wk 2a discussion – Inflammatory bowel disease
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